clsm proprietary image analysis program lsm software 2.3 (Carl Zeiss)
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Carl Zeiss
clsm proprietary image analysis program lsm software 2.3
Clsm Proprietary Image Analysis Program Lsm Software 2.3, supplied by Carl Zeiss, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/proprietary+image+analysis+software+program/axiovision+software/pm22198001-77-19-24
Average 90 stars, based on 1 article reviews
Clsm Proprietary Image Analysis Program Lsm Software 2.3, supplied by Carl Zeiss, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/proprietary+image+analysis+software+program/axiovision+software/pm22198001-77-19-24
Average 90 stars, based on 1 article reviews
clsm proprietary image analysis program lsm software 2.3 - by Bioz Stars,
2026-11
90/100 stars
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Confocal Laser Scanning Microscopy:Article Title: Protective role of hydrogen peroxide in oxygen-deprived dopaminergic neurones of the rat substantia nigra Article Snippet: .. Cellular labelling was analysed off-line through the Article Title: Axotomy dependent purinergic and nitrergic co-expression. Article Snippet: Different lines of evidence indicate that ATP and nitric oxide (NO) play key roles in mediating neuronal responses after cell damage.. Purinergic and nitrergic interactions have been proposed in non neural tissues physiological functions and, in different experimental models of brain injury, both purinergic and nitrergic activations have been reported.. The present study was planned to ascertain possible relations of these two systems after brain damage. Article Title: Distinct regulation of nNOS and iNOS by CB2 receptor in remote delayed neurodegeneration. Article Snippet: Hemicerebellectomy results in remote delayed degeneration of precerebellar neurons.. We have reported that such a lesion induces type 2 cannabinoid receptor (CB2) expression in precerebellar neurons and that stimulation of CB2, but not CB1, has neuroprotective effects.. In this study, we found that in the same model, the CB2 agonist JWH-015 enhances neuronal nitric oxide synthase (nNOS) expression in axotomized neurons and that CB2-mediated neuroprotection is abrogated by pharmacological inhibition of nNOS. Software:Article Title: Protective role of hydrogen peroxide in oxygen-deprived dopaminergic neurones of the rat substantia nigra Article Snippet: .. Cellular labelling was analysed off-line through the Article Title: Axotomy dependent purinergic and nitrergic co-expression. Article Snippet: Different lines of evidence indicate that ATP and nitric oxide (NO) play key roles in mediating neuronal responses after cell damage.. Purinergic and nitrergic interactions have been proposed in non neural tissues physiological functions and, in different experimental models of brain injury, both purinergic and nitrergic activations have been reported.. The present study was planned to ascertain possible relations of these two systems after brain damage. Article Title: Distinct regulation of nNOS and iNOS by CB2 receptor in remote delayed neurodegeneration. Article Snippet: Hemicerebellectomy results in remote delayed degeneration of precerebellar neurons.. We have reported that such a lesion induces type 2 cannabinoid receptor (CB2) expression in precerebellar neurons and that stimulation of CB2, but not CB1, has neuroprotective effects.. In this study, we found that in the same model, the CB2 agonist JWH-015 enhances neuronal nitric oxide synthase (nNOS) expression in axotomized neurons and that CB2-mediated neuroprotection is abrogated by pharmacological inhibition of nNOS. Immunolabeling:Article Title: Axotomy dependent purinergic and nitrergic co-expression. Article Snippet: Different lines of evidence indicate that ATP and nitric oxide (NO) play key roles in mediating neuronal responses after cell damage.. Purinergic and nitrergic interactions have been proposed in non neural tissues physiological functions and, in different experimental models of brain injury, both purinergic and nitrergic activations have been reported.. The present study was planned to ascertain possible relations of these two systems after brain damage. Article Title: Distinct regulation of nNOS and iNOS by CB2 receptor in remote delayed neurodegeneration. Article Snippet: Hemicerebellectomy results in remote delayed degeneration of precerebellar neurons.. We have reported that such a lesion induces type 2 cannabinoid receptor (CB2) expression in precerebellar neurons and that stimulation of CB2, but not CB1, has neuroprotective effects.. In this study, we found that in the same model, the CB2 agonist JWH-015 enhances neuronal nitric oxide synthase (nNOS) expression in axotomized neurons and that CB2-mediated neuroprotection is abrogated by pharmacological inhibition of nNOS. Labeling:Article Title: Axotomy dependent purinergic and nitrergic co-expression. Article Snippet: Different lines of evidence indicate that ATP and nitric oxide (NO) play key roles in mediating neuronal responses after cell damage.. Purinergic and nitrergic interactions have been proposed in non neural tissues physiological functions and, in different experimental models of brain injury, both purinergic and nitrergic activations have been reported.. The present study was planned to ascertain possible relations of these two systems after brain damage. Article Title: Distinct regulation of nNOS and iNOS by CB2 receptor in remote delayed neurodegeneration. Article Snippet: Hemicerebellectomy results in remote delayed degeneration of precerebellar neurons.. We have reported that such a lesion induces type 2 cannabinoid receptor (CB2) expression in precerebellar neurons and that stimulation of CB2, but not CB1, has neuroprotective effects.. In this study, we found that in the same model, the CB2 agonist JWH-015 enhances neuronal nitric oxide synthase (nNOS) expression in axotomized neurons and that CB2-mediated neuroprotection is abrogated by pharmacological inhibition of nNOS. |